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Marked suppression of dihydrotestosterone in men with benign prostatic hyperplasia by dutasteride, a dual 5α-Reductase inhibitor.

In a long-term efficacy and safety study, prostate volume was assessed yearly by magnetic resonance imaging (MRI) in a subset of patients.

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Leydig cells and an increase in serum LH levels (2- to 3-fold above control) has been demonstrated in both rodent species treated with high doses of Finasteride.

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Its actions on hair follicles, skin and prostate tissue is not direct however; These tissues are responsive to a form of testosterone called dihydrotestosterone (DHT); testosterone is converted to DHT by the enzyme 5-alpha-reductase.

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During a 4 to 6 year placebo- and comparator-controlled study that enrolled 3047 men, there were 4 cases of breast cancer in men treated with finasteride but no cases in men not treated with finasteride.

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PCPT: Evidence That Finasteride Reduces Risk of Most Frequently Detected Intermediate- and High-grade (Gleason Score 6 and 7) Cancer.

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Increases of about 10% were observed in luteinizing hormone (LH) and follicle-stimulating hormone (FSH) in patients receiving finasteride tablets, but levels remained within the normal range.

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Until further information is available, I recommend only the FDA-approved use of finasteride in men with severe symptoms from benign prostatic enlargement that have not responded to alpha-blocker therapy (such as Flomax) and want to delay surgical intervention for relief of urinary obstruction.

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However, finasteride does not reduce DHT levels to castrate levels since circulating testosterone is converted to DHT by the type 1 isoenzyme existing in the skin and liver.

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